Para grass, amphibians and reptiles

Exotic weeds invade ecosystems and cause reductions in plant and animal biodiversity, alteration of food chains, reduction of species abundances and alteration of assemblage structure of existing flora and faunal communities. These negative effects can severely reduce the ecosystem services provided by a previously valuable area, and important functions of natural ecosystems (such as maintenance of water quality, pollinator services, and maintenance of local biodiversity) can be compromised.

Among the invasive weeds most likely to have negative effects on ecosystems are grasses, because they compete successfully with native species in a wide range of ecosystem types, and can alter critical ecosystem processes, such as fire-proneness and nutrient cycling, of the invaded ecosystem. Introduced pasture grasses comprise a third of the worst environmental weeds in Australia.

Para grass has become established in waterways where it causes economic and ecological problems, such as flow reduction and increases in sedimentation, as well as reduction in the attractiveness of wetlands to water birds, and reduction in fish diversity. Evidence from Australian wetlands invaded by para grass suggests that it turns diverse wetland plant communities into a monoculture, drastically reducing both floral and faunal diversity. These changes are unlikely to be reversed simply by removing vegetation. In this case, vegetation removal on its own may simply degrade habitat further, rather than enhance it.

In this study, we examined the influence of para grass control, on amphibians and reptiles in a northern Australian wetland – the Townsville Town Common Conservation Park. We also examined uninvaded wetland areas adjacent to the para-grass-affected areas, to quantify the reptile and amphibian assemblages present in uninvaded wetlands. Finally, we observed the habitat use of an abundant predator (keelback snakes) to predict the responses of native fauna to the removal of the weed, and to understand some of the mechanisms responsible for these responses.